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Highlights
● Alterations in N-acetyl aspartate in acute stages and choline/creatine ratio in the acute stages of traumatic brain
injury are associated with post-traumatic neurocognitive disorders.
● Increased myo-inositol levels in subacute phases suggest significant neuronal damages, following traumatic brain
injury. The detection of lactate levels presents poorer outcomes.
● As more powerful magnetic resonance imaging scanners are available, the interest in extrametabolites, such as
glutamate, glutamine, and glutathione is on the rise; some studies reported significant associations between these metabolites
and PTND.